By W. Dean Wallace, Bita V. Naini
This atlas describes and illustrates the pathology of stable organ transplants and comprises chapters masking transplant pathology of the kidney, lung, center, liver, pancreas, small bowel and limbs. every one of those chapters in short experiences the most recent rejection classifications for every organ process with explanatory notes highlighting diagnostic standards, and tables directory entities within the differential analysis. integrated with each one bankruptcy are pictures demonstrating the pathology of the commonest and critical illnesses, in particular styles of organ rejection and similar entities or mimickers. Examples of vintage affliction strategies are supplied, in addition to ordinary displays which may spotlight and forestall diagnostic pitfalls.
A finished clarification of the immunogenetics of transplant rejection is incorporated, permitting the final pathologist to get to grips with an important features of serologic trying out during this sufferer inhabitants. A assessment of post-transplant lymphoproliferative illnesses is usually provided.
Practical Atlas of Transplant Pathology is geared toward either basic and specialist pathologists who come upon transplant pathology specimens of their practice.
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Extra info for Practical Atlas of Transplant Pathology
C. D. V. C. Fishbein b Fig. 1 Contraction-band artifact. Longitudinal (a) and cross-sectional (b) views of myocytes with contraction-band artifact. 1 Acute Cell-Mediated Rejection (Figs. 5) When cardiac transplantation was in its infancy, approximately 50 % of patients developed a hemodynamically significant episode of rejection during the first year after transplantation. Currently, at our institution, hemodynamically significant ACR occurs in less than 5 % of patients. As with other solid organs, ACR in the transplanted heart is mediated by host cytotoxic and helper T-cells targeting graft antigens.
In addition, it is also critical to establish a pre-transplant base level of non– HLA antibodies to understand the relevance of these antibodies to graft rejection. Continued efforts to define the non–HLA alloantigens and tissue-specific autoantigens involved in transplant rejection are essential for understanding the mechanisms and pathogenesis of non–HLA antibodies and development of treatment options. 1 Histocompatibility and Immunogenetics for Solid Organ Transplantation FITC-anti-human F(ab’)2 Non-HLA antigen 23 Median channel shift (MCS) Anti-endothelial cell antibody Surrogate mature endothelial cell or Tie2+ precursor endothelial cell Measure FITC intensity by flow cytometry Fig.
Three common formats of solid-phase tests for anti-HLA antibodies are in use today. Screening tests for anti-HLA antibody include class I and class II HLA purified separately from several individuals that cover a broad spectrum of HLA antigens. These tests provide a positive or negative result but do not yield information on the specificity of the antibody. Panel-reactive antibody (PRA) tests have an HLA class I or class II phenotype from one individual attached to each bead. These tests provide a percent PRA result (the percentage of positive reactions) and can be used to assign antibody specificity depending on the breadth of antibodies present in the serum.